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10.1038/s41580-020-00324-8 16 KaganV
Fallon RF, Goodenough DA
These results were further supported by another study from the same lab, whereby GLP-1 was shown to stimulate myocardial glucose uptake via mechanisms that were p38 MAP kinase and NO dependent, but independent of adenylyl cyclase or Akt [73]
A change in GSH levels is important when assessing toxicological responses and an indicator of oxidative stress, potentially leading to apoptosis or cell death
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