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This necessary opening creates a natural point of weakness
This Fc domain serves a dual function: it sterically hinders access to DPP-4, thereby protecting the GLP-1 moiety from enzymatic degradation, and it engages the neonatal Fc receptor (FcRn), facilitating receptor-mediated recycling and preventing lysosomal degradation following cellular uptake
GLP-1s arent typically liver-toxic and are being studied for improvement with fatty liver disease (MASLD/MASH)
This is a database analysis, not a clinical outcome study
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