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Thus, GLP-1R G-protein-biased ligands may offer new and unappreciated advantages in the context of chronic treatment such as promoting adipocyte hyperplasia, restoring insulin responsiveness and long-term glycaemic control while preserving pancreatic -cell function by minimizing the insulin secretory burden
In T2DM, there is reduced prandial release of GLP-1 with preserved pancreatic response , making GLP-1 an attractive pathway for antihyperglycaemic agents
doi: 10.3390/ijms141021021 Armogida M, Nistic R, Mercuri NB
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