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PMID 11095574.Free full text Lee, William M

Because typical early atherosclerotic lesion formation observed in hyperhomocysteinemic animals can be prevented by an antiplatelet therapy with dipyridamole, the authors concluded that platelet-mediated proliferation of smooth muscle cells is a potential cellular mechanism involved in the atherogenicity of hyperhomocysteinemia

PMID: 47 HurrellBPSakanoYShenSHelouDGLiMShafiei-JahaniPet al

It functions not only as a substrate for protein synthesis but also as a signaling molecule, predominantly through the activation of the mTORC1, which serves as a crucial regulator of MPS