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CNS GIPR KO mice resist development of diet-induced obesity To generate CNS-specific GIPR KO mice, Gipr fl/fl mice 17 were crossed to synapsin-Cre mice (Jackson Laboratories) to obtain Gipr fl/fl control mice and Gipr Syn/ mice (breeding schematic in Fig

-lipoic acid interaction with dopamine D2 receptor-dependent activation of the Akt/GSK-3 signaling pathway induced by antipsychotics: potential relevance for the treatment of schizophrenia

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Graphical Abstract Keywords Metabolic dysfunction-associated fatty liver disease, Nonalcoholic fatty liver disease, Nonalcoholic steatohepatitis, Pharmacological treatment Introduction Metabolic dysfunction-associated fatty liver disease (MAFLD) is a new term from nonalcoholic fatty liver disease (NAFLD), which is the positive-criteria diagnosis focusing on metabolic factors and independent of alcohol use