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Preclinical investigations encompassing ethanol-induced gastric mucosal lesions, NSAID-mediated ulceration, cysteamine-induced duodenal injury, and inflammatory bowel disease analogues have reported associations between BPC157 exposure and reductions in mucosal lesion surface area, preservation of submucosal vascular architecture, and modulation of mucosal structural integrity
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The day before the experiment, 2,000 cells/well were seeded in 96-well plates (Corning)
doi: 10.1038/S41596-021-00560-Y 205 PagliucaFWMillmanJRGrtlerMSegelMVan DervortARyuJHet al
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