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This immune exhaustion would lead to a decrease in the formation of memory T cells and an increase in the generation of short-lived effector cells and exhausted T cells, where these exhausted antiviral T cells, unable to become activated, are functionally ineffective and unable to exert their cytotoxic functions in response to antigenic stimulation [227, 229], resulting in impaired immunosurveillance of SARS-CoV-2-infected cells and other cells with latency to another virus, such as EBV [128, 139]

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