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PTL can target cancer cells via numerous mechanisms, such as inhibition of nuclear factor ()B, p53 activation and ROS stress.76 However, the mechanism of PTL toxicity to T-ALL has not been defined
Inhibiting glucagon release: GLP-1 prevents the release of glucagon (a hormone that raises blood sugar) from the pancreas, helping to reduce blood sugar levels
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In particular, USP22 raised GSH content and reduced the occurrence of ferroptosis in cardiomyocytes by activating the Sirt1-p53/SLC7A11 axis [252]
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