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The two are structurally distinct compounds commonly investigated together in tissue-related research contexts
The ASM is activated by several cellular stresses implicated in both apoptosis and autophagy by modulating the balance between ceramide and its metabolic derivatives, sphingosine (Sph) and sphingosine-1-phosphate (S1P) (Lee et al., 2014)
Studies on GHK-Cu include topical skin research, diabetic wound-healing research, cell studies, animal models, and formulation studies [2], [3], [14], [15]
They work at different biological layers: GHK-Cu at the dermis and ECM level, retinol at the epidermis and receptor level
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