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Chen D, Jin D, Huang S, Wu J, Xu M, Liu T, et al
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Here we show that HGF adopts an enhanced form with dynamically increased receptor-binding affinity and nitration-dysfunction resistance through interaction with lipoic acid trisulfide (LASSS) under physiological conditions
( GAP1 expression from its native promoter would not serve as a good control here because pGAP1 is repressed by the ammonium sulphate N-source and so D-HIS would not be taken up by the cells)
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