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Liraglutide reduces postprandial hyperlipidemia by increasing apob48 (apolipoprotein b48) catabolism and by reducing apob48 production in patients with type 2 diabetes mellitus

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have found that even at non-physiologically high action potential stimulations, inhibition of glycolysis or the mitochondrial ATP synthesis leads to only mild changes in presynaptic ATP levels [41]

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