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First, an amino acid substitution stops DPP-4 from destroying GLP-1
the dose of PT-141 may be such that a given patient is not therapeutically responsive to such dose, such as an IN dose of 7.5 mg in a patient responsive only to higher doses, such as 15 or 20 mg of PT-141
IGF-1 LR3 is hypothesized to mitigate these risks by supporting neuronal survival and improved cardiac output
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