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The induction of Bdnf mRNA under GIN treatment was partially prevented by FK506 or mitogen-activated protein kinase/extracellular signal-regulated protein kinase kinase 1/2 (MEK1/2) inhibitor U0126, respectively (Fig
2024 Jul;359:142252
Conclusion: Altogether, our results show that the cellular antioxidant defense systems contribute to leukemia resistance to MTX, and targeting these pathways, especially the thioredoxin antioxidant system, may be a promising strategy for resensitizing ALL to MTX
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