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Pharmacologically this yields a highly selective GHS-R1a agonist which, in classical rodent studies (Raun et al., Eur J Endocrinol 1998), does not significantly elevate cortisolemia, ACTH, prolactin or FSH/LH at effective doses, unlike GHRP-2 and GHRP-6
doi: 10.1128/IAI.69.11.6912-6922.2001 162 KroegelC.YukawaT.WestwickJ.BarnesP
10.1016/S0009-9120(87)80090-5
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