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J Nutr Ther 7(2):3139
By briefly blocking the OGF receptor, LDN appears to upregulate this entire pathway, supporting immune surveillance and, in some cases, limiting progression of inflammatory or immune-mediated concerns
ATP depletion secondary to mitochondrial dysfunction further limits PMCA turnover, establishing a reciprocal cycle in which impaired extrusion promotes mitochondrial Ca 2+ overload, which in turn worsens energetic insufficiency and ROS production (Millington-Burgess and Harper, 2021)
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