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This process was reported to also be active in epithelial cells under T2 conditions, in association with 15LO1 (27)
doi:10.1016/S0730-725X(99)00110-1
In addition, our results suggest a novel mechanism involving low GSH-associated AMPK activation that prevents alcohol-induced steatosis, which challenges the existing knowledge on the role of low GSH in alcoholic liver disease
The link between oxidative stress and disease pathogenesis is well-studied
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