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The key anti-inflammatory effects included: Modulation of macrophage activation states Decreased reactive nitrogen species production Reduced efferocytotic activity in treated cells Lower expression of pro-inflammatory mediators Research on corneal injuries found TB-500 suppressed multiple inflammatory mediators while maintaining bacterial clearance when combined with antibiotics
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"The promise of N -acetylcysteine in the treatment of obsessive-compulsive disorder"
![PDF] Resistance is not futile: gliotoxin biosynthesis, functionality and utility](https://figures.semanticscholar.org/d953f87324c0b9375a3223a5c1492bfc5c829db3/2-Figure1-1.png)
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