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We further explored the mechanism underlying the effect of GLP-1 treatment on preventing autophagy of endothelial cells and found that H 2 O 2 promoted autophagy in HUVECs cells through activation of the ROS dependent Akt and ERK1/2 signaling pathway
Bifidobacterium longum with fructo-oligosaccharides in patients with non alcoholic steatohepatitis
Among the 657,057 patients who received GLP-1 RAs, 231,075 received semaglutide, and 189,103 received dulaglutide
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