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Research indicates that the senescence-associated secretory phenotype (SASP) suppresses local stem cell activity and promotes a pro-inflammatory microenvironment hostile to regeneration in aging tissues
These strategies may involve diverse mechanisms in glial cells and neurons, including inhibition of apoptosis, reduction of excitotoxicity, suppression of neuroinflammation, enhancement of mitochondrial function, or promotion of neuronal regeneration [565, 566]
Yet, symptomatology is highly variable across individuals, shaped by underlying neurobiological factors
Nature 451 , 10691075 (2008)
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