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Combined Amylin/GLP-1 pharmacotherapy to promote and sustain long-lasting weight loss
10.1016/S0140-6736(64)90011-X 6
Physiology Mol
Reviews of the disease support the broad picture, describing MASH as a metabolically driven process in which excess fatty acids and toxic lipid intermediates can promote hepatocyte stress, macrophage activation and fibrotic signaling
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