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Generally, no
Mitochondrial Respiration Stress-Protective Hypoxia Effects DSIP has been shown to exert direct effects on brain mitochondrial respiration significantly increasing ADP-stimulated (state 3) mitochondrial respiration activity under conditions of experimental hypoxia
However, it may be inherited or acquired, with risk factors including hypertension, arrhythmias, valvular diseases, endocarditis, and metabolic disorders such as diabetes mellitus, Gaucher disease, and mitochondrial dysfunction (5, 6)
Presented for research literature review only Sorokin P (2011)
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