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In addition, it showed GSH-dependent high cytotoxicity on account of the rapid exhaust of intracellular GSH by both GSH depletion and biosynthesis inhibition
The established ability of GLP-1RAs to suppress glial cell activation and modulate cytokine releasekey drivers of these very pathwayspositions them as a uniquely targeted therapeutic strategy
Inhibition of mammalian target of rapamycin or apoptotic pathway induces autophagy and radiosensitizes PTEN null prostate cancer cells
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