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When cardiolipin becomes oxidized, it migrates to the outer membrane, where it can interact with cytoplasmic amyloidogenic proteins to facilitate the formation of pores in the mitochondrial membrane, and, ultimately, to trap mitochondrial debris from aged mitochondria within fibrillar extracellular deposits such as Lewy bodies (Seneff et al., 2025b)
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Kupfer dient dabei als essenzieller Cofaktor fr die Enzyme Lysyloxidase und Lysylhydroxylase, die fr die Quervernetzung und Stabilitt von Kollagenfasern verantwortlich sind
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