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Because of this rapid enzymatic degradation, native oral glutathione was found to have a baseline bioavailability of less than 1%, and a plasma half-life of roughly two minutes

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For example, when investigating the hypothesis that gap junctions transport toxic metabolites to neighboring cells and support their killing during APAP and thioacetamide hepatotoxicity, the fact that the new gap junction inhibitor 2-aminoethoxydipenyl-borate (2APB) protected against the injury appeared to support the hypothesis [154]