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What is missing is long-term, randomized, outcome-based clinical trial evidence for therapeutic uses
The potential mechanism of GLPs action in IBD may include promotion of tissue repair of injured epithelium, regulation of T cell differentiation and functions, regulation of innate immune cells such as macrophages and dendric cells, and reduction of proinflammatory cytokines (Fig
GLP-1s should not be used by individuals with a personal or family history of medullary thyroid carcinoma or Multiple Endocrine Neoplasia syndrome type 2 (MEN 2)
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