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ARA-290's interaction with the heightened EPOR-cR complex typically upregulated in response to cell or tissue damage, is also of interest
Presented for research literature review only Elssner T et al
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5 Conclusion Cisplatin is a highly effective and broad-spectrum anticancer drug, but its severe ototoxicity limits its clinical application, and its mechanism involves various pathways such as DNA damage, oxidative stress, inflammatory response, mitochondrial dysfunction, and ferroptosis
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