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Why myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS) may kill you: disorders in the inflammatory and oxidative and nitrosative stress (IO&NS) pathways may explain cardiovascular disorders in ME/CFS
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GLUCOSE-DEPENDENT INSULIN SECRETION A critical feature of GIP agonism is that it stimulates insulin secretion primarily when blood glucose is elevateda glucose-dependent mechanism that reduces hypoglycemia risk compared to compounds that stimulate insulin release independent of glucose levels
This results in a burst of ROS and lipid peroxidation, ultimately inducing the ferroptosis-like bacterial cell death ( S
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