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However, in glutathione synthetase deficiency, because the activity of gamma-glutamyl-cysteine synthetase is regulated via feedback inhibition by glutathione, gamma-glutamyl-cysteine and then L-5-oxoproline are overproduced, resulting in the metabolic acidosis and urinary excretion of this compound at levels several hundred-fold above normal (71

Integration performance was quantitatively evaluated using the integration Local Inverse Simpsons Index (iLISI)

Using BPC-157 beyond the active repair phase, once tissue remodeling has consolidated, offers diminishing returns by this model