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Longer-duration randomized controlled trials with hard clinical endpoints including liver-related morbidity and mortality are essential to definitively establish the clinical value of incretin-based therapies in MASH
Conclusions Ischemic stroke triggers a coordinated metabolic crisis characterized by ATP depletion, mitochondrial dysfunction, excitotoxicity, oxidative and nitrosative stress, maladaptive glycolysis, lactate and pH dysregulation, NAD + /FAD imbalance, inflammatory immunometabolism, endothelial-pericyte uncoupling, and impaired neurovascular repair
DMEM supplemented with 10% fetal bovine serum (FBS) and 1% penicillin-streptomycin was used for culture
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