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In human pulmonary fibroblast cultures, Epithalon treatment induced telomerase activity in cells that had ceased expressing the enzyme, resulting in telomere elongation and an extension of replicative lifespan beyond the Hayflick limit [1]

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These statements regarding detoxification support reflect glutathione's known biochemical functions but should not be interpreted as medical claims

The exact cause involves a combination of genetic predisposition, autoimmune attacks, and oxidative stress