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Concentrations decline with age
The contribution of oxidation in PD pathogenesis has also been supported through clinical studies reporting alterations in serum glutathione peroxidase and superoxide dismutase activities, together with high malondialdehyde (MDA) levels in PD patients 23,24
At the molecular level, astaxanthin upregulates synaptic proteins involved in both pre- and postsynaptic function, such as synaptophysin, PSD-95, and SNAP-25, while promoting neurotrophic signaling through the BDNF/TrkBCREB pathway
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