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There is no direct evidence of umami stimulation and GLP-1 secretion, but the T1R1 receptors were co-expressed with GLP-1-expressing STC-1 cells (Reference Wang, Murthy and Grider80) , which suggests that umami receptors play a role in GLP-1 signalling
On the contrary, Acetyl-CoA is an activator of pyruvate carboxylase, thus promoting the TCA flux or the gluconeogenesis (Pietrocola et al., 2015)
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