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Because acetaminophen-induced renal injury is caused by oxidative stress, inflammation, and cellular apoptosis, it is expected that feselol, with its antioxidant and anti-inflammatory effects, regulates the production pathways of apoptosis-promoting molecules such as Bax and caspase-3, and also affects the expression of anti-apoptotic genes such as Bcl-2

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This was the first study to show that statins inhibit T H 1-cell development and promote T H 2-cell development and attenuate both acute and relapse phases of EAE