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The main mechanism of cardioprotective effects of GLP-1R agonists against oxidative stress-induced injury in H9c2 cells (cardiomyoblasts from Rattus norvegicus) and in cardiomyocytes is related to the scavenging of reactive oxygen species, by increasing the concentrations of endogenous antioxidant defenses and inhibiting cardiomyocyte apoptosis [42]

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Around 20% of GLP-1 users report reduced alcohol consumption since starting the medication, 6 and FTI Consulting analysis estimates that adoption could cut cumulative U.S