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In conditions like pyridoxine-dependent epilepsy or isoniazid toxicity, pyridoxine administration replenishes PLP stores, thereby restoring normal neurotransmitter levels and terminating seizures

It pushes fibroblasts to produce more Type I collagen, the strong organized stuff in a healthy tendon, rather than the weaker scar-tissue Type III

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The dosing regimen varies depending on whether neurological symptoms are present: With neurological involvement: 1 mg intramuscularly on alternate days until no further improvement, then 1 mg every 2 months [3] [4] Without neurological involvement: 1 mg intramuscularly three times a week for 2 weeks, then 1 mg every 2-3 months for maintenance [3] [4] There is some evidence that high-dose oral B12 (1000-2000 micrograms daily) may be effective even in some malabsorption conditions, though UK practice still generally favours intramuscular treatment in these cases