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Moreover, SIN inhibits inflammasome activation during the initiation of CNS inflammation, targeting the NLRP3 inflammasome via an AMPK-dependent pathway and modulating astrocyte dopamine D2 receptors and the Alpha B-crystallin (CRYAB)/STAT3 axis to exert neuroprotective effects (97)
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9, A) revealed that FITC-labeled DNZ-NLPs have diffused into the brain parenchyma, likely due to an increase in BBB permeability
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