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Additionally, GOT1, which is upregulated in exosomes from patients with PDAC, promotes the expression of its binding protein CCR2 and upregulates both Nrf2 and HO-1, contributing to ferroptosis resistance in patients with PDAC [111]
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DPP-4 inhibitors The DPP-4 inhibitors mediate their action by inhibiting the enzyme DPP-4, which prevents the breakdown of GLP-1 and thereby increases endogenous GLP-1 levels
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