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How should healthcare providers approach the risk-benefit analysis for these treatments
There is no established pharmacologic mechanism by which GLP-1 receptor agonism would directly cause musculoskeletal pain
Depletion of GSH could lead to inactivation of SAM synthetase, with further decrease in GSH concentrations
Yes, FDA-approved versions of tirzepatidesuch as Mounjaro or Zepboundshould be considered as an alternative option
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