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We hypothesise that the absence of glucagon leads to improved oral glucose tolerance similar to that seen in glucagon receptor knockout mice, which also have minimal glucose excursions after an oral glucose load
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Appetite suppression also makes nutrition basics protein, fiber, water harder to hit, which is why dedicated GLP-1 trackers pair food logging with dose context rather than treating meals as a separate project
This is consistent across the GLP-1 class, as the STEP UP trial showed dramatically higher dysesthesia rates at higher semaglutide doses
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