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The anxiolytic mechanism involves GABA system modulation, enhancing inhibitory neurotransmission that counteracts excessive anxiety-driven neural activity

IL-17A causes depression-like symptoms via NFB and p38MAPK signaling pathways in mice: Implications for psoriasis associated depression

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Structurally, AOD 9604 retains a disulfide-linked cyclic conformation formed by cysteine residues, preserving key features of the native GH region responsible for lipid metabolism signaling