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2013 Jan 11;288(2):837-47
In multiple sclerosis models, GLP-1 receptor activation reduces pathogenic Th1 and Th17 cell activity, inhibits microglial activation (the brain equivalent of macrophage inflammation), and may enhance remyelination processes
Of the group, 88% were using a GLP-1 receptor agonist for the first time
Acid-sensing ion channel 1a (ASIC1a) is a key mediator of RA-associated joint cartilage destruction
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