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E103242
Slow intestinal transit may prolong contact with biliary metabolites and increase opportunities for microbial deconjugation and enterohepatic reabsorption
mild: (025%), moderate: (26-50%), good: (51-75%), excellent: (76100%) Patients were followed-up for three months after the end of treatment period
Notably, PFK158 significantly reduced glucose uptake and lactic acid production, and promoted atherosclerotic plaque stability [34], highlighting its anti-atherosclerosis potential
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