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MOTS-c modulates the FOXO transcription factors and regulates cellular senescence, the state in which aged or damaged cells stop dividing but refuse to die, instead secreting a cocktail of inflammatory signals known as the senescence-associated secretory phenotype (SASP)

This response depends on costimulatory signals and is tightly regulated by inhibitory immune checkpoints such as cytotoxic T-lymphocyte antigen-4 (CTLA-4) to prevent autoimmunity [18, 19]

For a more editorial, shorter read on the same protocol, see my piece on drworldwide.com: After the Operating Room: The Peptide Stack My Patients Recover On

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