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Because PDI bound by 4-HNE was identified in vivo, where GSH is present, it suggests that this compensatory mechanism may be limited by high levels of oxidative stress in which GSH concentrations are low [91]
This sophisticated transport system effectively balances iron utilization and storage while preventing iron-mediated oxidative damage ( Figure 1 )
and (3) severe: hemolytic anemia, metabolic acidosis, and progressive neurologic symptoms (62)
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