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This is illustrated by a study reporting that Lp(a) was associated with thrombosis [28], stenosis, and occlusion, but not plaque burden [29]
Rather than indiscriminately blocking macrophage activity (which would impair the early inflammatory debridement phase of wound healing), GHK-Cu research documents a shift in macrophage phenotype from pro-inflammatory M1 toward the anti-inflammatory, pro-healing M2 profile at later stages of the inflammatory response
It is not approved by the FDA for human or veterinary use
Its development is driven by the growing body of evidence linking receptor co-activation to enhanced therapeutic efficacy in the management of conditions such as obesity, type 2 diabetes, and metabolic dysfunction
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