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By activating GLP-1, GIP, and glucagon receptors simultaneously, it produces a distinct metabolic effect profile that combines the appetite and insulin effects of GLP-1/GIP activation with the energy-expenditure effects of glucagon receptor activation
What he and others are studying is how GLP-1 produced in the brain and GLP-1 produced in the gut interact, which could inform whether the effects of semaglutide in reducing alcohol drinking are related to the effects of semaglutide in the body
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