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While redox homeostasis via enhanced glutathione production is commonly associated with the normal physiological role of NRF2, collectively our results suggest that NRF2-dependent cancer cell growth does not require this enhanced glutathione production
Non-covalent NRF2 activation confers greater cellular protection than covalent activation
The page displays the current USCL 070626 COA, HPLC, and MS PDFs
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