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Conversely, FUNDC1-mediated mitophagy, once dephosphorylated, mainly exerts protective effects on the heart by removing damaged mitochondria, reducing ROS production, and inhibiting mPTP opening, thereby improving outcomes in myocardial ischaemiareperfusion injury [135, 136]
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It operates through two distinct receptor pathways that affect digestion, blood sugar regulation, and satiety signaling in ways that are fundamentally different from older weight loss medications
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