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There is no clinical data supporting combination use
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At the molecular level, AOD-9604 interacts with beta-3 adrenergic receptors on adipocytes (fat cells), triggering a signaling cascade that increases intracellular cyclic AMP levels
These mechanisms are worth mentioning since if the results in this latest correspondence in the NEJM are indeed replicated, there is a large difference if these effects are occurring either a) Because pancreatic beta cells are dying less b) Because pancreatic beta cells are growing back c) Because pancreatic beta cells are not being targeted by the immune system anymore If replicated, I find a) and b) to be the most likely reasons
