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Targeting inflammasomes could offer a potential therapeutic approach to mitigate neuroinflammation and slow the progression of PD

This suggests that the total levels of the TCA cycle intermediates are also decreased, which was corroborated in the forebrain of mice during the chronic stage of the pilocarpine-SE model (Willis et al., 2010), but not in hippocampus (McDonald et al., 2017)

This demonstrates how a gut-focused LDN and peptide combination can have profound effects on neurological symptoms linked to gut-brain axis dysfunction

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